About Adoptee Rage

Statistics Identify large populations of Adoptees in prisons, mental hospitals and committed suicide.
Fifty years of scientific studies on child adoption resulting in psychological harm to the child and
poor outcomes for a child's future.
Medical and psychological attempts to heal the broken bonds of adoption, promote reunions of biological parents and adult children. The other half of attempting to repair a severed Identity is counselling therapy to rebuild the self.

Saturday, September 21, 2013

Adopted Child Lack Of Attachment Result

ADOPTEE RAGE!

Adopted Children's Lack Of Attachment Result
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Primary attachment to help modulate stress

There is evidence that caring and secure environments help to moderate the negative impact that stress places on the developing brain (Gunnar, 1998). Because safety and bonding are crucial factors in the early construction of the brain, childhood trauma compromises core neural networks (Cozolino, 2002).
Normal play and exploratory activity in children requires the presence of a familiar attachment figure who can help modulate each child's physiological arousal by providing a balance between soothing and stimulation (Streeck-Fischer & van der Kolk, 2000). Children feel secure when the caregiver provides consistent, warm and sensitive care (Davila & Levy, 2006). In secure environments, stressed children after seeking and receiving comfort from their primary caregiver return to their exploratory activity away from the primary caregiver (p. 989). The caregiver's appropriate soothing response not only protects the child from the effects of stressful situations but it also enables the child to develop the biological framework for dealing with future stress (Schore, 1994; cited in Streeck-Fischer & van der Kolk, 2000). Acquiring controllable stress reactions seems to result in central nervous system reactions that facilitate the capacity to deal with subsequent stresses.
Devoid of a secure base, children find they cannot rely on the primary caregiver for comfort and may become incapable of calming themselves down when threatened. In addition, if children are exposed to unmanageable stress and the caregiver does not help modulate the child's arousal (as in situations of family violence) the child will be unable to organise his/her experiences in a coherent fashion (Streeck-Fischer & van der Kolk, 2000). If the child cannot regulate his/her emotional states, or rely on others to help: he/she will respond with fight or flight reactions. Cognitive understanding of events helps modulate emotions and enables the formulation of a flexible response. Both cognition and emotions are important. Children who are denied parental care or comfort for long periods of time, can suffer extreme mental and emotional deficits (Van Der Horst, LeRoy, & Van Der Veer, 2008).
Our first intimate or loving relationship is with our primary caregiver and this informs our expectations and patterns of behaviour (Harlow, 1958). For example, Bowlby (1969; 1973; 1980; 1988) identified a strong relationship between the pattern of attachment in young children and the patterns of their intimate relationships in later life. The negative core schema adopted by a survivor as a result of that first attachment fundamentally affects that survivor's capacity to establish and sustain significant attachments throughout life. Survivors often experience conflictual relationships and chaotic lifestyles, frequently report difficulties forming adult intimate attachments and display behaviours that threaten and disrupt close relationships (Henderson, 2006).

We have all heard the saying "What doesn't kill you makes you stronger" and "time heals all wounds". These bits of common wisdom conjure a picture of traumatic experiences that, once overcome, result in greater levels of psychological, physical and emotional wellbeing. Although trials and tribulations can certainly build character, they can also create permanent biological, neurological and psychological compromise (Cozolino, 2002). The impact of traumatic events on infants and young children is often minimized in this way. It is ironic that during infancy and childhood, a time of the greatest vulnerability to the effects of trauma, adults generally presume greater resilience (Perry, Pollard, Blakely, Baker, & Vigilante, 1995). The effects of early and severe trauma are widespread, devastating and difficult to treat (Cozolino, 2002; Giarratano, 2004a).
Childhood trauma can cause severe disturbances in the integration of sensory, emotional and cognitive information into a cohesive whole. This sets the stage for unfocused and irrelevant responses to subsequent stress (Cozolino, 2002; Streeck-Fischer & van der Kolk, 2000). For example, a lack of capacity for emotional self-regulation has been commonly observed in children who experience abuse and neglect (Streeck-Fischer & van der Kolk, 2000). Childhood trauma has profound impact on the emotional, behavioural, cognitive, social and physical functioning of children (Perry et al., 1995). Among other impacts, a traumatised child may, over time, exhibit motor hyperactivity, anxiety, behavioural impulsivity, sleep problems, and hypertension (Perry et al., 1995).
Similarly, adult survivors of childhood abuse have consistently identified impairments in adult physical and mental health in studies (Draper et al., 2007). Even though not everyone exhibits the same set of symptoms, or experiences the same intensity of problems, research shows that the long-term effects of child abuse are pervasive, across all areas of a survivor's world.
Although the association between childhood abuse and adult mental and physical health problems is well documented, less is known about the pathways through which health is compromised. An understanding of the effects of child abuse and neglect on a child's physical (biological), cognitive, social, behavioural and emotional development helps make sense of their repercussions in adulthood. Pathways linking childhood abuse with adult health outcomes span emotional, behavioural, social, cognitive, and biological pathways.